Prenatal Exposure Delayed Effects
"Prenatal Exposure Delayed Effects" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The consequences of exposing the FETUS in utero to certain factors, such as NUTRITION PHYSIOLOGICAL PHENOMENA; PHYSIOLOGICAL STRESS; DRUGS; RADIATION; and other physical or chemical factors. These consequences are observed later in the offspring after BIRTH.
| Descriptor ID |
D011297
|
| MeSH Number(s) |
C13.703.824.500
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Prenatal Exposure Delayed Effects".
Below are MeSH descriptors whose meaning is more specific than "Prenatal Exposure Delayed Effects".
This graph shows the total number of publications written about "Prenatal Exposure Delayed Effects" by people in this website by year, and whether "Prenatal Exposure Delayed Effects" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2001 | 0 | 1 | 1 |
| 2003 | 1 | 1 | 2 |
| 2004 | 3 | 0 | 3 |
| 2005 | 1 | 1 | 2 |
| 2007 | 5 | 0 | 5 |
| 2008 | 0 | 2 | 2 |
| 2009 | 1 | 1 | 2 |
| 2010 | 3 | 2 | 5 |
| 2011 | 2 | 1 | 3 |
| 2012 | 3 | 1 | 4 |
| 2013 | 5 | 2 | 7 |
| 2014 | 4 | 0 | 4 |
| 2015 | 2 | 2 | 4 |
| 2016 | 2 | 0 | 2 |
| 2017 | 1 | 1 | 2 |
| 2018 | 3 | 1 | 4 |
| 2019 | 3 | 0 | 3 |
| 2020 | 8 | 1 | 9 |
| 2021 | 3 | 0 | 3 |
| 2022 | 1 | 0 | 1 |
| 2023 | 1 | 0 | 1 |
| 2024 | 3 | 2 | 5 |
| 2026 | 2 | 0 | 2 |
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Below are the most recent publications written about "Prenatal Exposure Delayed Effects" by people in Profiles.
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Maternal undernutrition in pregnancy and lactation programs impaired immune system development and function in male offspring. J Dev Orig Health Dis. 2026 Apr 01; 17:e18.
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Perinatal Nicotine Exposure-Induced Cardiac Phenotype Is Mediated by miR-1-MIAT Interaction and Is Transmitted Transgenerationally. FASEB J. 2026 Mar 31; 40(6):e71711.
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Epigenetic Upregulation of Carotid Body Angiotensin Signaling Increases Blood Pressure. Hypertension. 2025 Feb; 82(2):293-305.
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Maternal e-cigarette exposure alters DNA methylome, site-specific CpG and CH methylation, and transcriptomic signatures in the neonatal brain. Sci Rep. 2024 10 16; 14(1):24263.
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Perinatal nicotine vaping exposure induces pro-myofibroblastic phenotype in rat bone marrow-derived mesenchymal stem cells. Reprod Toxicol. 2024 10; 129:108673.
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In utero chronic intermittent nicotine aerosol exposure increases ischemic heart injury in adult offspring via programming of Angiotensin II receptor-derived TGF?/ROS/Akt signaling pathway. Reprod Toxicol. 2024 09; 128:108650.
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Sex-Dependent Variations in Hypothalamic Fatty Acid Profile and Neuropeptides in Offspring Exposed to Maternal Obesity and High-Fat Diet. Nutrients. 2024 Jan 24; 16(3).
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BIOMARKERS OF MATERNAL SMOKING AND THE RISK OF RETINOBLASTOMA IN OFFSPRING. Retina. 2023 03 01; 43(3):481-489.
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Perinatal Exposure to Nicotine Alters Sperm RNA Profiles in Rats. Front Endocrinol (Lausanne). 2022; 13:893863.
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Fetal e-cigarette exposure programs a neonatal brain hypoxic-ischemic sensitive phenotype via altering DNA methylation patterns and autophagy signaling pathway. Am J Physiol Regul Integr Comp Physiol. 2021 11 01; 321(5):R791-R801.