Antigens, Differentiation
"Antigens, Differentiation" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Antigens expressed primarily on the membranes of living cells during sequential stages of maturation and differentiation. As immunologic markers they have high organ and tissue specificity and are useful as probes in studies of normal cell development as well as neoplastic transformation.
Descriptor ID |
D000943
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MeSH Number(s) |
D23.050.301.264 D23.101.100
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Concept/Terms |
Antigens, Differentiation- Antigens, Differentiation
- Differentiation Markers
- Markers, Differentiation
- Differentiation Antigens
- Marker Antigens
- Antigens, Marker
|
Below are MeSH descriptors whose meaning is more general than "Antigens, Differentiation".
Below are MeSH descriptors whose meaning is more specific than "Antigens, Differentiation".
This graph shows the total number of publications written about "Antigens, Differentiation" by people in this website by year, and whether "Antigens, Differentiation" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2001 | 2 | 0 | 2 |
2004 | 1 | 1 | 2 |
2005 | 1 | 0 | 1 |
2019 | 1 | 0 | 1 |
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Below are the most recent publications written about "Antigens, Differentiation" by people in Profiles.
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Vitrification and storage of oral mucosa epithelial cell sheets. J Tissue Eng Regen Med. 2019 07; 13(7):1153-1163.
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Contribution of Toll-like receptor/myeloid differentiation factor 88 signaling to murine liver regeneration. Hepatology. 2005 Mar; 41(3):443-50.
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ASC is essential for LPS-induced activation of procaspase-1 independently of TLR-associated signal adaptor molecules. Genes Cells. 2004 Nov; 9(11):1055-67.
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Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E. Proc Natl Acad Sci U S A. 2004 Jul 20; 101(29):10679-84.
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Plasmodium berghei infection in mice induces liver injury by an IL-12- and toll-like receptor/myeloid differentiation factor 88-dependent mechanism. J Immunol. 2001 Nov 15; 167(10):5928-34.
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Lipopolysaccharide-induced IL-18 secretion from murine Kupffer cells independently of myeloid differentiation factor 88 that is critically involved in induction of production of IL-12 and IL-1beta. J Immunol. 2001 Feb 15; 166(4):2651-7.