"Receptors, Immunologic" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cell surface molecules on cells of the immune system that specifically bind surface molecules or messenger molecules and trigger changes in the behavior of cells. Although these receptors were first identified in the immune system, many have important functions elsewhere.
Descriptor ID |
D011971
|
MeSH Number(s) |
D12.776.543.750.705
|
Concept/Terms |
Receptors, Immunologic- Receptors, Immunologic
- Immunologic Receptors
- Immunological Receptors
- Receptors, Immunological
- Immunologic Receptor
- Receptor, Immunologic
|
Below are MeSH descriptors whose meaning is more general than "Receptors, Immunologic".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Immunologic".
- Receptors, Immunologic
- Costimulatory and Inhibitory T-Cell Receptors
- Immunophilins
- Integrins
- Leukocyte Immunoglobulin-like Receptor B1
- Lipopolysaccharide Receptors
- Platelet Membrane Glycoproteins
- Receptors, Antigen
- Receptors, Complement
- Receptors, Cytokine
- Receptors, Fc
- Receptors, Formyl Peptide
- Receptors, Laminin
- Receptors, Lymphocyte Homing
- Receptors, Mitogen
- Receptors, Natural Killer Cell
- Receptors, Pattern Recognition
- Receptors, Scavenger
- Signaling Lymphocytic Activation Molecule Family
- Triggering Receptor Expressed on Myeloid Cells-1
This graph shows the total number of publications written about "Receptors, Immunologic" by people in this website by year, and whether "Receptors, Immunologic" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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1997 | 1 | 0 | 1 |
2001 | 2 | 0 | 2 |
2004 | 1 | 1 | 2 |
2005 | 1 | 0 | 1 |
2012 | 0 | 1 | 1 |
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Below are the most recent publications written about "Receptors, Immunologic" by people in Profiles.
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GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease. Neuron. 2013 Apr 24; 78(2):256-68.
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Dysregulated immunophenotypic attributes of plasmacytoid but not myeloid dendritic cells in HIV-1 infected individuals in the absence of highly active anti-retroviral therapy. Clin Exp Immunol. 2012 Nov; 170(2):212-21.
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Contribution of Toll-like receptor/myeloid differentiation factor 88 signaling to murine liver regeneration. Hepatology. 2005 Mar; 41(3):443-50.
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ASC is essential for LPS-induced activation of procaspase-1 independently of TLR-associated signal adaptor molecules. Genes Cells. 2004 Nov; 9(11):1055-67.
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Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E. Proc Natl Acad Sci U S A. 2004 Jul 20; 101(29):10679-84.
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Plasmodium berghei infection in mice induces liver injury by an IL-12- and toll-like receptor/myeloid differentiation factor 88-dependent mechanism. J Immunol. 2001 Nov 15; 167(10):5928-34.
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Lipopolysaccharide-induced IL-18 secretion from murine Kupffer cells independently of myeloid differentiation factor 88 that is critically involved in induction of production of IL-12 and IL-1beta. J Immunol. 2001 Feb 15; 166(4):2651-7.
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Amyloid-beta peptide-receptor for advanced glycation endproduct interaction elicits neuronal expression of macrophage-colony stimulating factor: a proinflammatory pathway in Alzheimer disease. Proc Natl Acad Sci U S A. 1997 May 13; 94(10):5296-301.